Make Ur Own Coenzyme Q10

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Chlorophyll is the green pigment that makes green leaves green. If you search for chlorophyll in the medical literature, a lot of what you find is about fecal fluorescence, a way to detect the contamination of carcasses in the slaughterhouse with feces to reduce the risk of food poisoning from pathogens harbored within animal feces. Fecal matter gets on meat either “with knife entry through the hide into the carcass, and also splash back and aerosol [airborne] deposition of fecal matter during hide removal”—that is, when they’re peeling off the skin. If, however, the animals have been eating grass, you can pick up the poo with a black light. As you can see in my video How to Regenerate Coenzyme Q10 (CoQ10) Naturally, a solution of chlorophyll is green, but, under a UV light, it lights up as red. So, if you have a black light in a chicken slaughter plant, you can get a drop on the droppings. The problem is most chickens aren’t outside anymore. They’re no longer pecking at grass so there’s less fecal fluorescence. We could let them run around outside or we could save money by just adding a chlorophyll supplement to their feed so we can better “identify areas of gut-spill contamination” on the meat.

The reason I was looking up chlorophyll was to follow-up on the data I presented in my Eating Green to Prevent Cancer video, which suggests that chlorophyll may be able to block carcinogens. I found a few in vitro studies on the potential anti-inflammatory effects of chlorophyll. After all, green leaves have long been used to treat inflammation, so anti-inflammatory properties of chlorophyll and their break-down products after digestion were put to the test. And, indeed, they may represent “valuable and abundantly available anti-inflammatory agents.” Maybe that’s one reason why cruciferous vegetables, like kale and collard greens, are associated with decreased markers of inflammation.

In a petri dish, for example, if you lay down a layer of arterial lining cells, more inflammatory immune cells stick to them after you stimulate them with a toxic substance. We can bring down that inflammation with the anti-inflammatory drug aspirin or, even more so, by just dripping on some chlorophyll. Perhaps that’s one of the reasons kale consumers appear to live longer lives.

As interesting as I found that study to be, this next study blew my mind. The most abundant energy source on this planet is sunlight. However, only plants are able to use it directly—or so we thought. After eating plants, animals have chlorophyll in them, too, so might we also be able to derive energy directly from sunlight? Well, first of all, light can’t get through our skin, right? Wrong. This was demonstrated by century-old science—and every kid who’s ever shined a flashlight through her or his fingers, showing that the red wavelengths do get through. In fact, if you step outside on a sunny day, there’s enough light penetrating your skull and going through to your brain that you could read a book in there. Okay, so our internal organs are bathed in sunlight, and when we eat green leafy vegetables, the absorbed chlorophyll in our body does actually appear to produce cellular energy. But, unless we eat so many greens we turn green ourselves, the energy produced is probably negligible.

However, light-activated chlorophyll inside our body may help regenerate Coenzyme Q10. CoQ10 is an antioxidant our body basically makes from scratch using the same enzyme we use to make cholesterol—that is, the same enzyme that’s blocked by cholesterol-lowering statin drugs. So, if CoQ10 production gets caught in the crossfire, then maybe that explains why statins increase our risk of diabetes—namely, by accidently also reducing CoQ10 levels in a friendly-fire type of event. Maybe that’s why statins can lead to muscle breakdown. Given that, should statin users take CoQ10 supplements? No, they should sufficiently improve their diets to stop taking drugs that muck with their biochemistry! By doing so—by eating more plant-based chlorophyll-rich diets—you may best maintain your levels of active CoQ10, also known as ubiquinol. “However, when ubiquinol is used as an antioxidant, it is oxidized to ubiquinone. To act as an effective antioxidant, the body must regenerate ubiquinol from ubiquinone,” perhaps by using dietary chlorophyll metabolites and light.

Researchers exposed some ubiquinone and chlorophyll metabolites to the kind of light that makes it into our bloodstream. Poof! CoQ10 was reborn. But, without the chlorophyll or the light, nothing happened. By going outside we get light and, if we’re eating our veggies, chlorophyll, so maybe that’s how we maintain such high levels of CoQ10 in our bloodstream. Perhaps this explains why dark green leafy vegetables are so good for us. We know sun exposure can be good for us and that eating greens can be good for us. “These benefits are commonly attributed to an increase in vitamin D from sunlight exposure and consumption of antioxidants from green vegetables”—but is it possible that these explanations might be incomplete

Chlorophyll is the green pigment that makes green leaves green. If one searches for chlorophyll in the medical literature, a lot of what you find is about fecal fluorescence, a way to detect the contamination of carcasses with feces in the slaughterhouse to reduce the risk of food poisoning from pathogens harbored within animal feces.

See, fecal matter gets on meat either with knife entry through the hide into the carcass, and also splash back and airborne deposition of fecal matter when they’re peeling off the skin. But if they’ve been eating grass, you can pick up the poo with a black light. Here’s a solution of chlorophyll. Under a UV light, though, chlorophyll lights up red. So, if you have a black light in a chicken slaughter plant, you can get a drop on the droppings. The problem is we don’t let chickens outside anymore. They’re no longer pecking at grass; so, there’s less fecal fluorescence. We could let them run around, or save money by just adding a chlorophyll supplement to their feed, so we can better identify areas of gut-spill contamination on the meat.

The reason I was looking up chlorophyll was to follow-up on the data I presented in my Eating Green to Prevent Cancer video, suggesting that chlorophyll may be able to block carcinogens. There were a few in vitro studies on the potential anti-inflammatory effects of chlorophyll. After all, green leaves have long been used to treat inflammation; so, anti-inflammatory properties of chlorophyll and these properties’ break-down products after digestion were put to the test. And indeed, they may represent valuable and abundantly available anti-inflammatory agents. Maybe that’s one reason why cruciferous veggies, like kale and collard greens, are associated with decreased markers of inflammation.

In a petri dish, for example, if you lay down a layer of arterial lining cells, this is how many inflammatory immune cells stick to them before, and after, you stimulate them with a toxic substance. We can bring that inflammation down, though, with the anti-inflammatory drug, aspirin, or even more by just dripping on some chlorophyll. Perhaps that’s one of the reasons kale consumers may live longer lives.

This is the study, though, that blew my mind. Sunlight is the most abundant energy source on this planet. So far, so good. However, only plants are really able to use sunlight directly, or so we thought. After eating plants, animals, too, may be able to derive energy directly from sunlight as well. What?! First of all, light can’t get through our skin, right? Wrong, as was demonstrated by century-old science—and any kid who’s ever shined a flashlight through their fingers; the red wavelengths do get through. In fact, if you step outside on a sunny day, there’s enough light going through to your brain, you could read a book in there. OK, so our internal organs are bathed in sunlight, and absorbed chlorophyll in the body does actually appear to produce cellular energy, but unless we eat so many greens we turn green ourselves, the energy produced is probably negligible.

However, light-activated chlorophyll in our body may help regenerate Coenzyme Q10. CoQ10 is an antioxidant our body basically makes from scratch using the same enzyme that our body uses to make cholesterol, the same enzyme that’s blocked by cholesterol-lowering statin drugs. So, if CoQ10 production gets caught in the crossfire, then maybe that explains why statins increase our risk of diabetes, by accidently also reducing CoQ10 levels. Maybe that’s why statins can lead to muscle breakdown. So, should statin users take CoQ10 supplements? No, they should improve their diets sufficiently to stop taking drugs that muck with their biochemistry. And by doing so, by eating more plant-based chlorophyll-rich diets, they may best maintain their levels of active CoQ10, also known as ubiquinol. However, when ubiquinol is used as an antioxidant, it is oxidized to ubiquinone. And for ubiquinol to act as an effective antioxidant again, the body must regenerate ubiquinol from ubiquinone, maybe using dietary chlorophyll metabolites and light.

They exposed some ubiquinone and chlorophyll metabolites to the kind of light that makes it into our bloodstream, and poof, CoQ10 was reborn, but without the chlorophyll, or without the light, nothing happened.  And look, we get light, we get chlorophyll if we’re eating our veggies. Maybe that’s how we maintain such high levels of CoQ10 in our bloodstream. Maybe that explains why dark green leafy vegetables are so good for us. We know sun can be good for us; we know greens can be good for us. These benefits are commonly attributed to an increase in vitamin D from sunlight exposure and all the antioxidants from green vegetables. But maybe these explanations might be incomplete.

This video has it all: a mind-blowing mechanism, practical applicability, and poop—what more could you want?

Eating Green to Prevent Cancer is the prior chlorophyll video I mentioned.

Interested in learning more about the potential downsides of cholesterol-lowering statin drugs? I’ve produced other videos on the topic, including Statin Cholesterol Drugs and Invasive Breast Cancer and The Actual Benefit of Diet vs. Drugs.

If you haven’t yet, you can subscribe to my videos for free by clicking here.

Why do people who eat more plants get less cancer? We’ve talked about some phytonutrients that can act as antioxidants to douse free radicals, how some can boost our liver’s own detoxifying enzymes, and some that even boost our DNA repair enzymes, to patch up any damage done. But 22 years ago, the interceptor molecule hypothesis was postulated. Serving as a first line of defense, interceptors bind to mutagens and carcinogens, and thereby block them from coming in contact with our DNA in the first place.

See, many carcinogens, shown here in blue, have a flat ring system narrow enough to slip into the spine of our DNA, causing mutations. But if some interceptor were able to glom onto the carcinogen first, it may no longer fit into our DNA. So the search was on, combing for the existence of carcinogen-binding molecules, and in 2007 we discovered one such amazing molecule was chlorophyll! The most ubiquitous plant pigment in the world, that which makes dark green leafy vegetables dark green.

In subsequent years, the ability of chlorophyll to “totally abolish” DNA damage of human cells exposed to carcinogens was documented in a petri dish. But what we really need to see is does it work in people. But you can’t just give people carcinogens—unless, you pay them enough. “Effects of Chlorophyll on Low-Dose Aflatoxin in Human Volunteers.”

They had people drink a solution of radioactive aflatoxin, the carcinogen that used to be a problem in peanut butter—with or without spinach chlorophyll. Here’s the big spike in their bloodstream of aflatoxin without spinach in their stomach, and this is with. Apparently, the chlorophyll bound to the aflatoxin and prevented its absorption into the bloodstream. “In sum, these studies provide substantial evidence that chlorophylls can strongly inhibit uptake of aflatoxins in the whole animal”—which, in this case, was us.

 

 

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