Alzheimer Protocol

PQQ is known to participate in mitochondrial biogenesis and can activate sirtuin-1, which is implicated in cellular genomic stability and longevity. In Alzheimer’s disease, reduced levels of brain sirtuin-1 correlate with accumulation of toxic beta-amyloid and tau proteins, and brain hypometabolism (partly attributable to loss of mitochondrial efficiency) contributes to cognitive decline. For these reasons, PQQ supplementation is likewise being considered as a therapy for improving neuronal energy dynamics in Alzheimer’s disease.

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